Low-dose tributyltin triggers human chondrocyte senescence and mouse articular cartilage aging
Author:
Funder
Ministry of Science and Technology, Taiwan
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Toxicology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00204-022-03407-x.pdf
Reference58 articles.
1. Anerillas C, Abdelmohsen K, Gorospe M (2020) Regulation of senescence traits by MAPKs. Geroscience 42(2):397–408. https://doi.org/10.1007/s11357-020-00183-3
2. Antizar-Ladislao B (2008) Environmental levels, toxicity and human exposure to tributyltin (TBT)-contaminated marine environment: a review. Environ Int 34(2):292–308. https://doi.org/10.1016/j.envint.2007.09.005
3. Baker AH, Watt J, Huang CK, Gerstenfeld LC, Schlezinger JJ (2015) Tributyltin engages multiple nuclear receptor pathways and suppresses osteogenesis in bone marrow multipotent stromal cells. Chem Res Toxicol 28(6):1156–1166. https://doi.org/10.1021/tx500433r
4. Baker AH, Wu TH, Bolt AM, Gerstenfeld LC, Mann KK, Schlezinger JJ (2017) From the cover: tributyltin alters the bone marrow microenvironment and suppresses B cell development. Toxicol Sci 158(1):63–75. https://doi.org/10.1093/toxsci/kfx067
5. Banu N, Tsuchiya, Sawada R (2009) Effects of Tin compounds on human chondrogenic activity in vitro. In: Ikura K et al (eds) Animal cell technology: basic & applied aspects, vol 15: pp 181–186. https://doi.org/10.1007/978-1-4020-9646-4_29
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