Effect of N-acetyltransferase 2 genetic polymorphism on 4,4′-methylenebis(2-chloroaniline)-induced genotoxicity and oxidative stress
Author:
Funder
NIH
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Toxicology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00204-023-03508-1.pdf
Reference38 articles.
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2. Bellamri M, Walmsley SJ, Brown C et al (2022) DNA damage and oxidative stress of tobacco smoke condensate in human bladder epithelial cells. Chem Res Toxicol. https://doi.org/10.1021/acs.chemrestox.2c00153
3. Bendaly J, Doll MA, Millner LM et al (2009) Differences between human slow N-acetyltransferase 2 alleles in levels of 4-aminobiphenyl-induced DNA adducts and mutations. Mutat Res 671(1–2):13–19.https://doi.org/10.1016/j.mrfmmm.2009.08.003
4. Bonnier F, Keating ME, Wrobel TP et al (2015) Cell viability assessment using the Alamar blue assay: a comparison of 2D and 3D cell culture models. Toxicol in Vitro 29(1):124–131. https://doi.org/10.1016/j.tiv.2014.09.014
5. da Silva J (2016) DNA damage induced by occupational and environmental exposure to miscellaneous chemicals. Mutation Res 770:170–182. https://doi.org/10.1016/j.mrrev.2016.02.002
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2. The effect of the rs1799931 G857A (G286E) polymorphism on N-acetyltransferase 2-mediated carcinogen metabolism and genotoxicity differs with heterocyclic amine exposure;Archives of Toxicology;2023-08-18
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