Maternal NO2 exposure induces cardiac hypertrophy in male offspring via ROS-HIF-1α transcriptional regulation and aberrant DNA methylation modification of Csx/Nkx2.5-Reference-Cited by-同舟云学术

Maternal NO2 exposure induces cardiac hypertrophy in male offspring via ROS-HIF-1α transcriptional regulation and aberrant DNA methylation modification of Csx/Nkx2.5

Published:2018-02-07 Issue:4 Volume:92 Page:1563-1579
ISSN:0340-5761
Container-title:Archives of Toxicology
language:en
Short-container-title:Arch Toxicol

Author:

Zhang Yingying,Yan Wei,Ji Xiaotong,Yue Huifeng,Li Guangke,Sang Nan

Funder

National Science Foundation of PR China

Research Project Supported by Shanxi Scholarship Council of China

Research Project for Young Sanjin Scholarship of Shanxi

Program for the Outstanding Innovative Teams of Higher Learning Institutions of Shanxi

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology,General Medicine

Link

http://link.springer.com/article/10.1007/s00204-018-2166-3/fulltext.html

Reference83 articles.

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3. Al-Attas OS, Hussain T, Ahmed M, Al-Daghri N, Mohammed AA, De Rosas E, Gambhir D, Sumague TS (2015) Ultrastructural changes, increased oxidative stress, inflammation, and altered cardiac hypertrophic gene expressions in heart tissues of rats exposed to incense smoke. Environ Sci Pollut Res Int 22(13):10083–10093

4. Aragon AC, Goens MB, Carbett E, Walker MK (2008) Perinatal 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure sensitizes offspring to angiotensin II-induced hypertension. Cardiovasc Toxicol 8(3):145–154

5. Bansal M, Kaushal N (2014) Oxidative stress mechanisms and their modulation. Springer, India

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