Applying the adverse outcome pathway concept for assessing non-monotonic dose responses: biphasic effect of bis(2-ethylhexyl) phthalate (DEHP) on testosterone levels-Reference-Cited by-同舟云学术

Applying the adverse outcome pathway concept for assessing non-monotonic dose responses: biphasic effect of bis(2-ethylhexyl) phthalate (DEHP) on testosterone levels

Published:2022-11-07 Issue:2 Volume:97 Page:313-327
ISSN:0340-5761
Container-title:Archives of Toxicology
language:en
Short-container-title:Arch Toxicol

Author:

Astuto M. C.^ORCID,Benford D.,Bodin L.,Cattaneo I.,Halldorsson T.,Schlatter J.,Sharpe R. M.,Tarazona J.,Younes M.

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00204-022-03409-9.pdf

Reference107 articles.

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2. Ahmad S, Khan MF, Parvez S, Akhtar M, Raisuddin S (2017) Molecular docking reveals the potential ofphthalate esters to inhibit the enzymes of the glucocorticoid biosynthesis pathway. J Appl Toxicol 37:265–277. https://doi.org/10.1002/jat.3355

3. Akingbemi BT, Ge RS, Klinefelter GR, Zirkin BR, Hardy MP (2004) Phthalate-induced Leydig cell hyperplasia isassociated with multiple endocrine disturbances. Proc National Acad Sci USA 101:775–780

4. Ammini AC, Sunderaraman PG, Gupta R, Karmarkar MG, Buckshee K, Ahuja MM (1992) Congenital adrenal hyperplasia among peripubertal girls with hyperandrogenism. Indian Pediatr 29:79–83

5. Andrade AJM, Grande SW, Talsness CE, Gericke C, Grote K, Golombiewski A, Sterner-Kock A, Chahoud I (2006a) A dose response study following in utero and lactational exposure to di-(2-ethylhexyl) phthalate (DEHP): reproductive effects on adult male offspring rats. Toxicology 228:85–97. https://doi.org/10.1016/j.tox.2006.08.020

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