Highlight report: toxicology of copper
Author:
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Toxicology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00204-015-1648-9.pdf
Reference36 articles.
1. Aigner E, Weiss G, Datz C (2015) Dysregulation of iron and copper homeostasis in nonalcoholic fatty liver. World J Hepatol 7(2):177–188. doi: 10.4254/wjh.v7.i2.177 (Review)
2. Balmer NV, Klima S, Rempel E, Ivanova VN, Kolde R, Weng MK, Meganathan K, Henry M, Sachinidis A, Berthold MR, Hengstler JG, Rahnenführer J, Waldmann T, Leist M (2014) From transient transcriptome responses to disturbed neurodevelopment: role of histone acetylation and methylation as epigenetic switch between reversible and irreversible drug effects. Arch Toxicol 88(7):1451–1468. doi: 10.1007/s00204-014-1279-6
3. Bento I, Carrondo MA, Lindley PF (2006) Reduction of dioxygen by enzymes containing copper. J Biol Inorg Chem 11(5):539–547 (Review)
4. Campos G, Schmidt-Heck W, Ghallab A, Rochlitz K, Pütter L, Medinas DB, Hetz C, Widera A, Cadenas C, Begher-Tibbe B, Reif R, Günther G, Sachinidis A, Hengstler JG, Godoy P (2014) The transcription factor CHOP, a central component of the transcriptional regulatory network induced upon CCl4 intoxication in mouse liver, is not a critical mediator of hepatotoxicity. Arch Toxicol 88(6):1267–1280. doi: 10.1007/s00204-014-1240-8
5. Engle TE (2011) Copper and lipid metabolism in beef cattle: a review. J Anim Sci 89(2):591–596. doi: 10.2527/jas.2010-3395 (Review)
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1. Disruption of intestinal structure, tight junction complex, immune response and microbiota after chronic exposure to copper in swamp eel (Monopterus albus);Fish & Shellfish Immunology;2023-12
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3. Induction of autophagy via the ROS-dependent AMPK-mTOR pathway protects copper-induced spermatogenesis disorder;Redox Biology;2022-02
4. Cu-induced spermatogenesis disease is related to oxidative stress-mediated germ cell apoptosis and DNA damage;Journal of Hazardous Materials;2021-08
5. TGF-β1-induced EMT activation via both Smad-dependent and MAPK signaling pathways in Cu-induced pulmonary fibrosis;Toxicology and Applied Pharmacology;2021-05
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