Initial autophagic protection switches to disruption of autophagic flux by lysosomal instability during cadmium stress accrual in renal NRK-52E cells-Reference-Cited by-同舟云学术

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Initial autophagic protection switches to disruption of autophagic flux by lysosomal instability during cadmium stress accrual in renal NRK-52E cells

Published:2017-03-20 Issue:10 Volume:91 Page:3225-3245
ISSN:0340-5761
Container-title:Archives of Toxicology
language:en
Short-container-title:Arch Toxicol

Author:

Lee W.-K.,Probst S.,Santoyo-Sánchez M. P.,Al-Hamdani W.,Diebels I.,von Sivers J.-K.,Kerek E.,Prenner E. J.,Thévenod F.

Funder

Deutsche Forschungsgemeinschaft

Max-Kade Foundation

Consejo Nacional de Ciencia y Tecnología

Canadian Network for Research and Innovation in Machining Technology, Natural Sciences and Engineering Research Council of Canada (CA)

PhD Program Universität Witten/Herdecke

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology,General Medicine

Link

http://link.springer.com/article/10.1007/s00204-017-1942-9/fulltext.html

Reference84 articles.

1. Abouhamed M, Gburek J, Liu W et al (2006) Divalent metal transporter 1 in the kidney proximal tubule is expressed in late endosomes/lysosomal membranes: implications for renal handling of protein-metal complexes. Am J Physiol Renal Physiol 290(6):F1525–F1533

2. Aits S, Jaattela M (2013) Lysosomal cell death at a glance. J Cell Sci 126(Pt 9):1905–1912. doi: 10.1242/jcs.091181

3. Akesson A, Lundh T, Vahter M et al (2005) Tubular and glomerular kidney effects in Swedish women with low environmental cadmium exposure. Environ Health Perspect 113(11):1627–1631

4. Aki T, Funakoshi T, Unuma K, Uemura K (2013) Impairment of autophagy: from hereditary disorder to drug intoxication. Toxicology 311(3):205–215. doi: 10.1016/j.tox.2013.07.001

5. Amoruso MA, Witz G, Goldstein BD (1987) Alteration of erythrocyte membrane fluidity by heavy metal cations. Toxicol Ind Health 3(1):135–144

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1. Insoluble HIFa protein aggregates by cadmium disrupt hypoxia-prolyl hydroxylase (PHD)-hypoxia inducible factor (HIFa) signaling in renal epithelial (NRK-52E) and interstitial (FAIK3-5) cells;BioMetals;2024-09-10

2. Epigenetic alterations in bioaccumulators of cadmium: Lessons from mammalian kidneys and plants;Environment International;2024-09

3. Blockage of Akt activation suppresses cadmium-induced renal tubular cellular damages through aggrephagy in HK-2 cells;Scientific Reports;2024-06-24

4. Effect of Ba(II), Eu(III), and U(VI) on rat NRK-52E and human HEK-293 kidney cells in vitro;Science of The Total Environment;2024-05

5. Distinct concentration-dependent oxidative stress profiles by cadmium in a rat kidney proximal tubule cell line;Archives of Toxicology;2024-01-30

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