Inhibitory effect of snake venom toxin on NF-κB activity prevents human cervical cancer cell growth via increase of death receptor 3 and 5 expression-Reference-Cited by-同舟云学术

Inhibitory effect of snake venom toxin on NF-κB activity prevents human cervical cancer cell growth via increase of death receptor 3 and 5 expression

Published:2014-11-23 Issue:2 Volume:90 Page:463-477
ISSN:0340-5761
Container-title:Archives of Toxicology
language:en
Short-container-title:Arch Toxicol

Author:

Lee Hye Lim,Park Mi Hee,Hong Ji Eun,Kim Dae Hwan,Kim Ji Young,Seo Hyen Ok,Han Sang-Bae,Yoon Joo Hee,Lee Won Hyoung,Song Ho Sueb,Lee Ji In,Lee Ung Soo,Song Min Jong,Hong Jin Tae

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00204-014-1393-5.pdf

Reference58 articles.

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3. Ballestrero A, Nencioni A, Boy D et al (2004) Tumor necrosis factor-related apoptosis-inducing ligand cooperates with anticancer drugs to overcome chemoresistance in antiapoptotic Bcl-2 family members expressing jurkat cells. Clin Cancer Res 10(4):1463–1470

4. Ban JO, Jung YS, Kim DH et al (2014) (E)-2, 4-Bis(p-hydroxyphenyl)-2-butenal inhibits tumor growth via suppression of NF-kappaB and induction of death receptor 6. Apoptosis Int J Program Cell Death 19(1):165–178. doi: 10.1007/s10495-013-0903-x

5. Barkett M, Gilmore TD (1999) Control of apoptosis by Rel/NF-kappaB transcription factors. Oncogene 18(49):6910–6924. doi: 10.1038/sj.onc.1203238

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