Over-expression of genes and proteins of ubiquitin specific peptidases (USPs) and proteasome subunits (PSs) in breast cancer tissue observed by the methods of RFDD-PCR and proteomics
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology
Link
http://link.springer.com/content/pdf/10.1007/s10549-006-9393-7.pdf
Reference43 articles.
1. Rape M, Reddy SK, Kirschner MW (2006) The processivity of multiubiquitination by the APC determines the order of substrate degradation. Cell 124:89–103
2. Doucet C, Gutierrez GJ, Lindon C et al (2005) Multiple phosphorylation events control mitotic degradation of the muscle transcription factor Myf5. BMC Biochem 6:27
3. Asher G, Tsvetkov P, Kahana C et al (2005) A mechanism of ubiquitin-independent proteasomal degradation of the tumor suppressors p53 and p73. Genes Dev 19:316–321
4. Dragnev KH, Pitha-Rowe I, Ma Y et al (2004) Specific chemopreventive agents trigger proteasomal degradation of G1 cyclins: implications for combination therapy. Clin Cancer Res 10:2570–2577
5. Rossi S, Loda M (2003) The role of the ubiquitination-proteasome pathway in breast cancer: use of mouse models for analyzing ubiquitination processes. Breast Cancer Res 5:16–22
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