Head-to-Head Comparison of CCN4, DNMT3A, PTPN11, and SPARC as Suppressors of Anti-tumor Immunity
Author:
Funder
National Cancer Institute
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,Modeling and Simulation
Link
https://link.springer.com/content/pdf/10.1007/s12195-023-00787-7.pdf
Reference34 articles.
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3. Garris, C. S., et al. Successful anti-PD-1 cancer immunotherapy requires T cell-dendritic cell crosstalk involving the cytokines IFN-γ and IL-12. Immunity. 49(6):1148-1161.e7, 2018. https://doi.org/10.1016/j.immuni.2018.09.024.
4. Kulkarni, Y. M., E. Chambers, A. J. R. McGray, J. S. Ware, J. L. Bramson, and D. J. Klinke. A quantitative systems approach to identify paracrine mechanisms that locally suppress immune response to Interleukin-12 in the B16 melanoma model. Integr. Biol. (United Kingdom). 4(8):925–936, 2012. https://doi.org/10.1039/c2ib20053h.
5. Wu, Y., W. Deng, E. C. McGinley, and D. J. Klinke. Melanoma exosomes deliver a complex biological payload that upregulates PTPN11 to suppress T lymphocyte function. Pigment Cell Melanoma Res. 30(2):203–218, 2017. https://doi.org/10.1111/pcmr.12564.
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