Downregulation of Major Histocompatibility Complex Class I Expression in Polymorphonuclear Leukocytes by Triggering CD 1 6 Antigen

Author:

Nishimura Motoko1,Ishikawa Yoshihide1,Mitsunaga Shigeki2

Affiliation:

1. Department of Research, Tokyo Metropolitan Red Cross, Blood Center, Tokyo, Japan

2. Department of Research, The Japanese Red Cross, Central Blood Center, Tokyo, Japan

Publisher

Japanese Society for Histocompatibility and Immunogenetics

Subject

General Medicine

Reference11 articles.

1. 1. Fanger NA, Lin C, Guyre PM, et al.: Activation of human T cells by major histocompatibility complex class II expressing neutrophil: Proliferation in the presence of super antigen, but not tetanus toxoid. Blood 89:4128-4135, 1997.

2. 2. Radska M, Iking-Konert S, Andrassy K, et al.: Polymorphonuclear nutrophils as accessory cells for T-cell activation: major histocompatibility complex class II restricted antigen-dependent induction of T cell proliferation. Immunology 101:521-530, 2000.

3. 3. Huizinga TWJ, Kleijer M, Tetterco PA, et al.: Biallelic neutrophil Na-antigen system is associated with a polymorphism on the phospho-inositol-linked Fe ァ receptor III (CD16). Blood 7 5: 213-217,1990.

4. 4. Huizinga TWJ, Kuijpers RW, Kleijer M, et al.: Maternal genomic neutrophil Fe’RIii deficiency leading to neonatal isoimmune neutropenia. Blood 76:1927-1932,1990.

5. 5. Stroncek DF, Skubiz KM, Plachta LB. et al.: Alloimmune neonatal neutropenia due to an antibody to the neutrophil Fe-’receptor III with maternal deficiency of CD16 antigen. Blood 77:1572-1580, 1991.

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