High-fat-diet-modulated Gut Microbiota Promotes Intestinal Carcinogenesis
Author:
Affiliation:
1. Department of Microbiology and Immunology, School of Medicine and Brain Korea 21 PLUS Program, and Institute of Medical Science, Jeju National University, Jeju, Korea.
Publisher
The Korean Society for Microbiology and The Korean Society of Virology
Subject
Virology,Immunology,Microbiology
Link
https://synapse.koreamed.org/pdf/10.4167/jbv.2015.45.4.394
Reference8 articles.
1. High-fat-diet-mediated dysbiosis promotes intestinal carcinogenesis independently of obesity
2. ERK activation drives intestinal tumorigenesis in Apcmin/+ mice
3. Microbiota-induced activation of epithelial IL-6 signaling links inflammasome-driven inflammation with transmissible cancer
4. Intestinal Inflammation Targets Cancer-Inducing Activity of the Microbiota
5. Activation of Gpr109a, Receptor for Niacin and the Commensal Metabolite Butyrate, Suppresses Colonic Inflammation and Carcinogenesis
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