Opportunity “Nox”: A Novel Approach to Preventing Endothelial Dysfunction in the Context of Insulin Resistance
Author:
Affiliation:
1. College of Health, University of Utah, Salt Lake City, Utah
2. Division of Endocrinology, Metabolism, and Diabetes, School of Medicine, University of Utah, Salt Lake City, Utah
Publisher
American Diabetes Association
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://journals.org/diabetes/diabetes/article-pdf/62/6/1818/570874/1818.pdf
Reference22 articles.
1. A review of endothelial dysfunction in diabetes: a focus on the contribution of a dysfunctional eNOS;Triggle;J Am Soc Hypertens,2010
2. Lipotoxicity contributes to endothelial dysfunction: A focus on the contribution from ceramide;Symons;Rev Endocr Metab Disord,2013;14:59–68
3. The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology;Bedard;Physiol Rev,2007
4. Combating oxidative stress in vascular disease: NADPH oxidases as therapeutic targets;Drummond;Nat Rev Drug Discov,2011
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1. Physiological Doses of Oleic and Palmitic Acids Protect Human Endothelial Cells from Oxidative Stress;Molecules;2022-08-16
2. A Genetic Polymorphism in the Pannexin1 Gene Predisposes for The Development of Endothelial Dysfunction with Increasing BMI;Biomolecules;2020-01-31
3. Blueberry Metabolites Attenuate Lipotoxicity-Induced Endothelial Dysfunction;Molecular Nutrition & Food Research;2017-12-14
4. Endothelial dysfunction — A major mediator of diabetic vascular disease;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2013-12
5. Response to Comment on: Sukumar et al. Nox2 NADPH Oxidase Has a Critical Role in Insulin Resistance–Related Endothelial Cell Dysfunction. Diabetes 2013;62:2130–2134;Diabetes;2013-11-16
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