Syntaxin 4 Enrichment in β-Cells Prevents Conversion to Autoimmune Diabetes in Non-Obese Diabetic (NOD) Mice
Author:
Publisher
American Diabetes Association
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Reference45 articles.
1. Type 1 diabetes
2. Type 1 diabetes mellitus as a disease of the β-cell (do not blame the immune system?);Roep;Nat Rev Endocrinol,2021
3. Reduced insulin secretion correlates with decreased expression of exocytotic genes in pancreatic islets from patients with type 2 diabetes
4. Impaired Gene and Protein Expression of Exocytotic Soluble N-Ethylmaleimide Attachment Protein Receptor Complex Proteins in Pancreatic Islets of Type 2 Diabetic Patients
5. Syntaxin 4 Expression in Pancreatic β-Cells Promotes Islet Function and Protects Functional β-Cell Mass
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Spontaneous Akt2 deficiency in a colony of NOD mice exhibiting early diabetes;Scientific Reports;2024-04-20
2. Syntaxin interacts with arachidonic acid to prevent diabetes mellitus;Lipids in Health and Disease;2022-08-18
3. Exocytosis Proteins: Typical and Atypical Mechanisms of Action in Skeletal Muscle;Frontiers in Endocrinology;2022-06-14
4. Syntaxin 4 — protective in β-cells in T1DM?;Nature Reviews Endocrinology;2021-10-18
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