Visuomotor Performance in KCNJ11-Related Neonatal Diabetes Is Impaired in Children With DEND-Associated Mutations and May Be Improved by Early Treatment With Sulfonylureas-Reference-Cited by-同舟云学术

Visuomotor Performance in KCNJ11-Related Neonatal Diabetes Is Impaired in Children With DEND-Associated Mutations and May Be Improved by Early Treatment With Sulfonylureas

Published:2012-08-01 Issue:10 Volume:35 Page:2086-2088
ISSN:0149-5992
Container-title:Diabetes Care
language:en
Short-container-title:Diabetes Care

Author:

Shah R. P.,Spruyt K.,Kragie B. C.,Greeley S. A. W.,Msall M. E.

Publisher

American Diabetes Association

Subject

Advanced and Specialised Nursing,Endocrinology, Diabetes and Metabolism,Internal Medicine

Reference14 articles.

1. Switching from Insulin to Oral Sulfonylureas in Patients with Diabetes Due to Kir6.2 Mutations

2. Activating Mutations in the Gene Encoding the ATP-Sensitive Potassium-Channel Subunit Kir6.2 and Permanent Neonatal Diabetes

3. Mutations in KCNJ11, which encodes Kir6.2, are a common cause of diabetes diagnosed in the first 6 months of life, with the phenotype determined by genotype

4. Muscle Dysfunction Caused by a K ATP Channel Mutation in Neonatal Diabetes Is Neuronal in Origin

5. Distribution and phenotype of neurons containing the ATP-sensitive K+ channel in rat brain

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