Adipocyte-Specific Deletion of Lamin A/C Largely Models Human Familial Partial Lipodystrophy Type 2
Author:
Affiliation:
1. Department of Molecular & Integrative Physiology, University of Michigan, Ann Arbor, MI
2. Division of Metabolism, Endocrinology and Diabetes, Department of Internal Medicine, University of Michigan, Ann Arbor, MI
Abstract
Publisher
American Diabetes Association
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://journals.org/diabetes/diabetes/article-pdf/70/9/1970/627962/db201001.pdf
Reference41 articles.
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3. Loss of A-type lamin expression compromises nuclear envelope integrity leading to muscular dystrophy;Sullivan;J Cell Biol,1999
4. Post-natal myogenic and adipogenic developmental: defects and metabolic impairment upon loss of A-type lamins;Kubben;Nucleus,2011
5. Deregulation of Fragile X-related protein 1 by the lipodystrophic lamin A p.R482W mutation elicits a myogenic gene expression program in preadipocytes;Oldenburg;Hum Mol Genet,2014
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