Update to Expression of Concern. Kathrin Maedler, Desiree M. Schumann, Nadine Sauter, Helga Ellingsgaard, Domenico Bosco, Reto Baertschiger, Yoichiro Iwakura, José Oberholzer, Claes B. Wollheim, Benoit R. Gauthier, and Marc Y. Donath. Low Concentration of Interleukin-1β Induces FLICE-Inhibitory Protein–Mediated β-Cell Proliferation in Human Pancreatic Islets. Diabetes 2006;55:2713-2722. DOI:10.2337/db05-1430. PMID: 17003335
Author:
Publisher
American Diabetes Association
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Reference7 articles.
1. Low Concentration of Interleukin-1β Induces FLICE-Inhibitory Protein–Mediated β-Cell Proliferation in Human Pancreatic Islets
2. Deletion of the Mitochondrial Flavoprotein Apoptosis Inducing Factor (AIF) Induces β-Cell Apoptosis and Impairs β-Cell Mass
3. Ardestani A , Sauter NS , Paroni F , . Neutralizing IL-1β induces β-cell survival by maintaining PDX1 protein nuclear localization [PAP version], 10 March 2011. J Biol Chem. Available from http://www.jbc.org/content/early/2011/03/10/jbc.M110.210526.full.pdf
4. Notice of correction of “Deletion of the mitochondrial flavoprotein apoptosis inducing factor (AIF) induces beta-cell apoptosis and impairs beta-cell mass.” PLoS One 2009;4:e4394. https://doi.org/10.1371/journal.pone.0117766
5. Notice of retraction of “Neutralizing interleukin-1β (IL-1β) induces β-cell survival by maintaining PDX1 protein nuclear localization” [retraction of: Ardestani A, Sauter NS, Paroni F, et al. In: J Biol Chem 2011;286:17144–17155]. J Biol Chem 2015;290:27532. https://doi.org/10.1074/jbc.A110.210526
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