Comment on: Butler et al. Marked Expansion of Exocrine and Endocrine Pancreas With Incretin Therapy in Humans With Increased Exocrine Pancreas Dysplasia and the Potential for Glucagon-Producing Neuroendocrine Tumors. Diabetes 2013;62:2595

Comment on: Butler et al. Marked Expansion of Exocrine and Endocrine Pancreas With Incretin Therapy in Humans With Increased Exocrine Pancreas Dysplasia and the Potential for Glucagon-Producing Neuroendocrine Tumors. Diabetes 2013;62:2595–2604

Published:2013-09-17 Issue:10 Volume:62 Page:e16-e17
ISSN:0012-1797
Container-title:Diabetes
language:en
Short-container-title:

Author:

Heine Robert J.¹,Fu Haoda¹,Kendall David M.¹,Moller David E.¹

Affiliation:

1. Lilly Research Laboratories and Lilly Diabetes, Eli Lilly & Co., Indianapolis, Indiana.

Publisher

American Diabetes Association

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://journals.org/diabetes/diabetes/article-pdf/62/10/e16/570528/e16.pdf

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2. Pancreatic fibrosis associated with age and ductal papillary hyperplasia;Detlefsen;Virchows Arch,2005

3. β-cell replication is increased in donor organs from young patients after prolonged life support;In’t Veld;Diabetes,2010

4. Chronic GLP-1 receptor activation by exendin-4 induces expansion of pancreatic duct glands in rats and accelerates formation of dysplastic lesions and chronic pancreatitis in the Kras(G12D) mouse model;Gier;Diabetes,2012

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