High prevalence of autoantibodies to glutamic acid decarboxylase in long-standing IDDM is not a marker of symptomatic autonomic neuropathy-Reference-Cited by-同舟云学术

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High prevalence of autoantibodies to glutamic acid decarboxylase in long-standing IDDM is not a marker of symptomatic autonomic neuropathy

Published:1994-09-01 Issue:9 Volume:43 Page:1146-1151
ISSN:0012-1797
Container-title:Diabetes
language:en
Short-container-title:Diabetes

Author:

Zanone M. M.,Petersen J. S.,Peakman M.,Mathias C. J.,Watkins P. J.,Dyrberg T.,Vergani D.

Publisher

American Diabetes Association

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Cited by 28 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Older age of childhood type 1 diabetes onset is associated with islet autoantibody positivity >30 years later: the Pittsburgh Epidemiology of Diabetes Complications Study;Diabetic Medicine;2020-02-11

2. GAD65 neurological autoimmunity;Muscle & Nerve;2017-04-11

3. Autoimmune-mediated peripheral neuropathies and autoimmune pain;Handbook of Clinical Neurology;2016

4. Prevalence of autoantibodies and risk estimation of development of youth onset type 1 diabetes in northern India;Diabetes & Metabolic Syndrome: Clinical Research & Reviews;2008-02

5. Antibodies to GAD65 and peripheral nerve function in the DCCT;Journal of Neuroimmunology;2007-04

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