New Tricks for Nrf2: Therapeutic Targeting to Restore BK-β1 Expression?-Reference-Cited by-同舟云学术

New Tricks for Nrf2: Therapeutic Targeting to Restore BK-β1 Expression?

Published:2017-09-13 Issue:10 Volume:66 Page:2538-2540
ISSN:0012-1797
Container-title:Diabetes
language:en
Short-container-title:

Author:

Chapple Sarah J.¹^ORCID,Mann Giovanni E.¹

Affiliation:

1. King’s British Heart Foundation Centre of Research Excellence, Cardiovascular Division, Faculty of Life Sciences & Medicine, King’s College London, London, U.K.

Funder

British Heart Foundation

Heart Research UK

Publisher

American Diabetes Association

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://journals.org/diabetes/diabetes/article-pdf/66/10/2538/534261/dbi170024.pdf

Reference25 articles.

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3. Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain;Itoh;Genes Dev,1999

4. Keap1 recruits Neh2 through binding to ETGE and DLG motifs: characterization of the two-site molecular recognition model;Tong;Mol Cell Biol,2006

5. Dimerization of substrate adaptors can facilitate cullin-mediated ubiquitylation of proteins by a “tethering” mechanism: a two-site interaction model for the Nrf2-Keap1 complex;McMahon;J Biol Chem,2006

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