ID’ing a Novel Inhibitor of β-Cell Function, Id1
Author:
Affiliation:
1. Department of Biomedical Sciences, University of Copenhagen, Copenhagen, Denmark
Publisher
American Diabetes Association
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://journals.org/diabetes/diabetes/article-pdf/60/10/2455/398262/2455.pdf
Reference15 articles.
1. Islet β cell failure in type 2 diabetes;Prentki;J Clin Invest,2006
2. The beta cell lesion in type 2 diabetes: there has to be a primary functional abnormality;Kahn;Diabetologia,2009
3. Chronic hyperglycemia, independent of plasma lipid levels, is sufficient for the loss of beta-cell differentiation and secretory function in the db/db mouse model of diabetes;Kjørholt;Diabetes,2005
4. Expression profiling of palmitate- and oleate-regulated genes provides novel insights into the effects of chronic lipid exposure on pancreatic β-cell function;Busch;Diabetes,2002
5. Glucose and other insulin secretagogues induce, rather than inhibit, expression of Id-1 and Id-3 in pancreatic islet beta cells;Wice;Diabetologia,2001
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