Use of a Dual GIP/GLP-1 Receptor Agonist in HNF1A-MODY and HNF4A-MODY
Author:
Affiliation:
1. 1Division of Internal Medicine, Department of Metabolism, Endocrinology & Diabetes, University of Michigan, Ann Arbor, MI
2. 2Department of Human Genetics, University of Michigan, Ann Arbor, MI
Publisher
American Diabetes Association
Link
https://diabetesjournals.org/care/article-pdf/47/9/e65/780465/dc240730.pdf
Reference5 articles.
1. Prevalence, characteristics and clinical diagnosis of maturity onset diabetes of the young due to mutations in HNF1A, HNF4A, and glucokinase: results from the SEARCH for Diabetes in Youth;Pihoker;J Clin Endocrinol Metab,2013
2. Maturity-onset diabetes of the young (MODY): how many cases are we missing?;Shields;Diabetalogia,2010
3. Glucose-lowering effects and low risk of hypoglycemia in patients with maturity-onset diabetes of the young when treated with a GLP-1 receptor agonist: a double-blind, randomized, crossover trial;Østoft;Diabetes Care,2014
4. GIP and GLP-1 potentiate sulfonylurea-induced insulin secretion in hepatocyte nuclear factor 1α mutation carriers;Christensen;Diabetes,2020
5. Tirzepatide once weekly for the treatment of obesity in people with type 2 diabetes (SURMOUNT-2): a double-blind, randomised, multicentre, placebo-controlled, phase 3 trial;Garvey;Lancet,2023
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