Defects in IL-2R Signaling Contribute to Diminished Maintenance of FOXP3 Expression in CD4+CD25+ Regulatory T-Cells of Type 1 Diabetic Subjects
Author:
Affiliation:
1. Benaroya Research Institute at Virginia Mason, Seattle, Washington;
2. Department of Biochemistry and Molecular Biology, Indiana University School of Medicine, Indianapolis, Indiana;
3. Seattle Children's Hospital, Seattle, Washington.
Abstract
Publisher
American Diabetes Association
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://journals.org/diabetes/diabetes/article-pdf/59/2/407/394910/zdb00210000407.pdf
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3. Interleukin-2 receptor signaling in regulatory T cell development and homeostasis;Burchill;Immunol Lett,2007
4. The biology of interleukin-2;Malek;Annu Rev Immunol,2008
5. IL-2 is essential for TGF-beta to convert naive CD4+CD25- cells to CD25+Foxp3+ regulatory T cells and for expansion of these cells;Zheng;J Immunol,2007
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