Neonatal exposure to sevoflurane caused learning and memory impairment via dysregulating SK2 channel endocytosis
Author:
Affiliation:
1. Xiangya School of Medicine, Central South University, China
2. Department of Anesthesiology, Guizhou Provincial People’s Hospital, The Affiliated Hospital of Guizhou University, China
3. Department of Biomedicine, Guizhou University, China
Abstract
Funder
National Natural Science Foundation of China
Guizhou Provincial Natural Science Foundation of China
Guizhou Provincial High-level creative talents cultivation plan: Thousand plan
Publisher
SAGE Publications
Subject
Multidisciplinary
Link
http://journals.sagepub.com/doi/pdf/10.1177/00368504211043763
Reference22 articles.
1. Anesthesia Neurotoxicity in the Developing Brain
2. Effects of sevoflurane on learning, memory, and expression of pERK1/2 in hippocampus in neonatal rats
3. Neonatal exposure to sevoflurane caused cognitive deficits by dysregulating SK2 channels and GluA2-lacking AMPA receptors in juvenile rat hippocampus
4. Developmental mapping of small-conductance calcium-activated potassium channel expression in the rat nervous system
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1. Melatonin attenuates sevoflurane-induced hippocampal damage and cognitive deficits in neonatal mice by suppressing CypD in parvalbumin neurons;Brain Research Bulletin;2023-11
2. Apamin, an SK2 Inhibitor, Attenuated Neonatal Sevoflurane Exposures Caused Cognitive Deficits in Mice through the Regulation of Hippocampal Neuroinflammation;ACS Chemical Neuroscience;2023-08-30
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