Aberrant V(D)J Recombination in Ataxia Telangiectasia Mutated-Deficient Lymphocytes Is Dependent on Nonhomologous DNA End Joining-Reference-Cited by-同舟云学术

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Aberrant V(D)J Recombination in Ataxia Telangiectasia Mutated-Deficient Lymphocytes Is Dependent on Nonhomologous DNA End Joining

Published:2008-08-06 Issue:4 Volume:181 Page:2620-2625
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Bredemeyer Andrea L.,Huang Ching-Yu,Walker Laura M.,Bassing Craig H.,Sleckman Barry P.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference30 articles.

1. Somatic generation of antibody diversity

2. The RAG Proteins and V(D)J Recombination: Complexes, Ends, and Transposition

3. The role of the non-homologous end-joining pathway in lymphocyte development

4. Cernunnos, a Novel Nonhomologous End-Joining Factor, Is Mutated in Human Immunodeficiency with Microcephaly

5. XLF Interacts with the XRCC4-DNA Ligase IV Complex to Promote DNA Nonhomologous End-Joining

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1. Unfolding the symbiosis of AID, chromatin remodelers, and epigenetics–The ACE phenomenon of antibody diversity;Immunology Letters;2024-10

2. ATM and 53BP1 regulate alternative end joining–mediated V(D)J recombination;Science Advances;2024-08-02

3. ATM and 53BP1 regulate alternative end joining-mediated V(D)J recombination;2024-04-25

4. DNA-PKcs suppresses illegitimate chromosome rearrangements;Nucleic Acids Research;2024-02-27

5. Inborn Errors of Immunity and Cytokine Storm Syndromes;Advances in Experimental Medicine and Biology;2024

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