Sex as a Profound Modifier of Atherosclerotic Lesion Development in Apolipoprotein E-deficient Mice with Different Genetic Backgrounds-Reference-Cited by-同舟云学术

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Sex as a Profound Modifier of Atherosclerotic Lesion Development in Apolipoprotein E-deficient Mice with Different Genetic Backgrounds

Published:2010 Issue:7 Volume:17 Page:712-721
ISSN:1880-3873
Container-title:Journal of Atherosclerosis and Thrombosis
language:en
Short-container-title:JAT

Author:

C. Surra Joaquín,Guillén Natalía,M. Arbonés-Mainar José,Barranquero Cristina,A. Navarro María,Arnal Carmen,Orman Israel,C. Segovia José,Osada Jesús

Publisher

Japan Atherosclerosis Society

Subject

Biochemistry (medical),Cardiology and Cardiovascular Medicine,Internal Medicine

Reference29 articles.

1. Variation in susceptibility to atherosclerosis among inbred strains of mice

2. Atherosclerosis and plasma and liver lipids in nine inbred strains of mice

3. Using Mice to Dissect Genetic Factors in Atherosclerosis

4. Understanding the role of dietary components on atherosclerosis using genetic engineered mouse models

5. 5) Shi W, Wang NJ, Shih DM, Sun VZ, Wang X, Lusis AJ: Determinants of atherosclerosis susceptibility in the C3H and C57BL/6 mouse model: evidence for involvement of endothelial cells but not blood cells or cholesterol metab-olism. Circ Res, 2000; 86: 1078-1084

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Comprehensive Analysis of 1-Year-Old Female Apolipoprotein E-Deficient Mice Reveals Advanced Atherosclerosis with Vulnerable Plaque Characteristics;International Journal of Molecular Sciences;2024-01-22

2. Iron Restriction Alleviates Atherosclerosis in ApoE KO Mice: An iTRAQ Proteomic Analysis;International Journal of Molecular Sciences;2022-12-14

3. Acetyl-CoA Deficiency Is Involved in the Regulation of Iron Overload on Lipid Metabolism in Apolipoprotein E Knockout Mice;Molecules;2022-08-04

4. TCDD aggravates the formation of the atherosclerotic plaque in ApoE KO mice with a sexual dimorphic pattern;Biochimie;2022-04

5. Maternal hypercholesterolemia exacerbates atherosclerosis lesions in female offspring through potentiating macrophage polarization toward an inflammatory M1 phenotype;The Journal of Nutritional Biochemistry;2021-04

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