Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population-Reference-Cited by-同舟云学术

Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population

Published:2010-05-03 Issue:5 Volume:120 Page:1674-1682
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Antonelli Lis R.V.,Gigliotti Rothfuchs Antonio,Gonçalves Ricardo,Roffê Ester,Cheever Allen W.,Bafica Andre,Salazar Andres M.,Feng Carl G.,Sher Alan

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference45 articles.

1. Virus interference. I. The interferon

2. Type 1 Interferon (IFNα/β) and Type 2 Nitric Oxide Synthase Regulate the Innate Immune Response to a Protozoan Parasite

3. Beta Interferon Is Produced by Chlamydia trachomatis -Infected Fibroblast-Like Synoviocytes and Inhibits Gamma Interferon-Induced HLA-DR Expression

4. The function of type I interferons in antimicrobial immunity

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