CCM3 signaling through sterile 20–like kinases plays an essential role during zebrafish cardiovascular development and cerebral cavernous malformations-Reference-Cited by-同舟云学术

CCM3 signaling through sterile 20–like kinases plays an essential role during zebrafish cardiovascular development and cerebral cavernous malformations

Published:2010-08-02 Issue:8 Volume:120 Page:2795-2804
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Zheng Xiangjian,Xu Chong,Di Lorenzo Annarita,Kleaveland Benjamin,Zou Zhiying,Seiler Christoph,Chen Mei,Cheng Lan,Xiao Jiping,He Jie,Pack Michael A.,Sessa William C.,Kahn Mark L.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference43 articles.

1. Cerebral Cavernous Malformations

2. Mutations in the Gene Encoding KRIT1, a Krev-1/rap1a Binding Protein, Cause Cerebral Cavernous Malformations (CCM1)

3. KRIT1 is mutated in hyperkeratotic cutaneous capillary-venous malformation associated with cerebral capillary malformation

4. Mutations in a Gene Encoding a Novel Protein Containing a Phosphotyrosine-Binding Domain Cause Type 2 Cerebral Cavernous Malformations

5. Mutations within the MGC4607 Gene Cause Cerebral Cavernous Malformations

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