MERTK inhibition alters the PD-1 axis and promotes anti-leukemia immunity-Reference-Cited by-同舟云学术

MERTK inhibition alters the PD-1 axis and promotes anti-leukemia immunity

Published:2018-11-02 Issue:21 Volume:3 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Lee-Sherick Alisa B.,Jacobsen Kristen M.,Henry Curtis J.,Huey Madeline G.,Parker Rebecca E.^ORCID,Page Lauren S.,Hill Amanda A.,Wang Xiaodong^ORCID,Frye Stephen V.,Earp H. Shelton,Jordan Craig T.,DeRyckere Deborah^ORCID,Graham Douglas K.

Funder

National Institutes of Health

Pablove Foundation

National Cancer Institute NCI NeXT program

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/97941/files/pdf

Reference44 articles.

1. Mer receptor tyrosine kinase is a therapeutic target in pre–B-cell acute lymphoblastic leukemia

2. Inhibition of MerTK increases chemosensitivity and decreases oncogenic potential in T-cell acute lymphoblastic leukemia;Brandao;Blood Cancer J,2013

3. UNC2025, a MERTK Small-Molecule Inhibitor, Is Therapeutically Effective Alone and in Combination with Methotrexate in Leukemia Models

4. Mertk Receptor Mutation Reduces Efferocytosis Efficiency and Promotes Apoptotic Cell Accumulation and Plaque Necrosis in Atherosclerotic Lesions of Apoe −/− Mice

5. A role for Mer tyrosine kinase in αvβ5 integrin-mediated phagocytosis of apoptotic cells;Wu;J Cell Sci,2005

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