Protein 4.1R–deficient mice are viable but have erythroid membrane skeleton abnormalities-Reference-Cited by-同舟云学术

Protein 4.1R–deficient mice are viable but have erythroid membrane skeleton abnormalities

Published:1999-02-01 Issue:3 Volume:103 Page:331-340
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Shi Zheng-Tao,Afzal Veena,Coller Barry,Patel Dipti,Chasis Joel A.,Parra Marilyn,Lee Gloria,Paszty Chris,Stevens Mary,Walensky Loren,Peters Luanne L.,Mohandas Narla,Rubin Edward,Conboy John G.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference45 articles.

1. Deficiency of skeletal membrane protein band 4.1 in homozygous hereditary elliptocytosis. Implications for erythrocyte membrane stability.

2. Restoration of normal membrane stability to unstable protein 4.1-deficient erythrocyte membranes by incorporation of purified protein 4.1.

3. Differentiation-associated switches in protein 4.1 expression. Synthesis of multiple structural isoforms during normal human erythropoiesis.

4. Tissue-specific analogues of erythrocyte protein 4.1 retain functional domains

5. Membrane skeletal protein 4.1 of avian erythrocytes is composed of multiple variants that exhibit tissue-specific expression

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