HIV protease inhibitors promote atherosclerotic lesion formation independent of dyslipidemia by increasing CD36-dependent cholesteryl ester accumulation in macrophages-Reference-Cited by-同舟云学术

HIV protease inhibitors promote atherosclerotic lesion formation independent of dyslipidemia by increasing CD36-dependent cholesteryl ester accumulation in macrophages

Published:2003-02-01 Issue:3 Volume:111 Page:389-397
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Dressman James,Kincer Jeanie,Matveev Sergey V.,Guo Ling,Greenberg Richard N.,Guerin Theresa,Meade David,Li Xiang-An,Zhu Weifei,Uittenbogaard Annette,Wilson Melinda E.,Smart Eric J.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference40 articles.

1. Atherogenic Dyslipidemia in HIV-Infected Individuals Treated With Protease Inhibitors

2. HIV Protease Inhibitor Induces Fatty Acid and Sterol Biosynthesis in Liver and Adipose Tissues Due to the Accumulation of Activated Sterol Regulatory Element-binding Proteins in the Nucleus

3. Replacing ritonavir by nelfinavir or nelfinavir/saquinavir as part of highly active antiretroviral therapy leads to an improvement of triglyceride levels

4. Dyslipidaemia in HIV-infected patients: association with adherence to potent antiretroviral therapy

5. Use of Human Immunodeficiency Virus-1 Protease Inhibitors Is Associated With Atherogenic Lipoprotein Changes and Endothelial Dysfunction

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