Effect of Human Papillomavirus on Cell Cycle–Related Proteins p16, Ki-67, Cyclin D1, p53, and ProEx C in Precursor Lesions of Cervical Carcinoma

Author:

Conesa-Zamora Pablo1,Doménech-Peris Asunción2,Orantes-Casado Francisco J.1,Ortiz-Reina Sebastián3,Sahuquillo-Frías Laura2,Acosta-Ortega Jesús3,García-Solano José3,Pérez-Guillermo Miguel3

Affiliation:

1. Pharmacogenetic and Molecular Pathology Group, Santa María del Rosell University Hospital, Cartagena, Spain

2. Clinical Analysis Department, Santa María del Rosell University Hospital, Cartagena, Spain

3. Pathology Department, Santa María del Rosell University Hospital, Cartagena, Spain

Publisher

Oxford University Press (OUP)

Subject

General Medicine

Reference37 articles.

1. Global cancer statistics;Parkin;CA Cancer J Clin,1999

2. Molecular pathogenesis of cancer of the cervix and its causation by specific human papillomavirus types;Zur Hausen;Curr Top Microbiol Immunol,1994

3. The expressions of the Rb pathway in cervical intraepithelial neoplasia: predictive and prognostic significance;Nam;Gynecol Oncol,2007

4. Overexpression of p16(INK4A) as a specific marker for dysplastic and neoplastic epithelial cells of the cervix uteri;Klaes;Int J Cancer,2001

5. Aberrant expression of cyclin D1 is associated with poor prognosis in early stage cervical cancer of the uterus;Bae;Gynecol Oncol,2001

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