Genetic and Biochemical Evidence That EBNA 2 Interaction with a 63-kDa Cellular GTG-Binding Protein Is Essential for B Lymphocyte Growth Transformation by EBV
Author:
Publisher
Elsevier BV
Subject
Virology
Cited by 81 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Regulation of B cell receptor signalling by Epstein–Barr virus nuclear antigens;Biochemical Journal;2022-12-07
2. EBNA2-EBF1 complexes promote MYC expression and metabolic processes driving S-phase progression of Epstein-Barr virus–infected B cells;Proceedings of the National Academy of Sciences;2022-07-20
3. The EBNA2-EBF1 complex promotes oncogenic MYC expression levels and metabolic processes required for cell cycle progression of Epstein-Barr virus-infected B cells;2021-12-29
4. The interplay between Epstein-Bar virus (EBV) with the p53 and its homologs during EBV associated malignancies;Heliyon;2019-11
5. Epstein–Barr Virus-Associated Malignancies: Roles of Viral Oncoproteins in Carcinogenesis;Frontiers in Oncology;2018-08-02
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