Mechanistic insights into the regulation of transcription and transcription-coupled DNA repair by Cockayne syndrome protein B

Author:

Boetefuer Erica L1,Lake Robert J2,Fan Hua-Ying2ORCID

Affiliation:

1. Department of Biology, University of Pennsylvania, Philadelphia, PA 19104, USA

2. Department of Internal Medicine, Division of Molecular Medicine, Program in Cancer Genetics, Epigenetics and Genomics, University of New Mexico Comprehensive Cancer Center, Albuquerque, NM 87131, USA

Funder

National Institutes of Health

Cancer Center support

Publisher

Oxford University Press (OUP)

Subject

Genetics

Reference78 articles.

1. Selective removal of transcription-blocking DNA damage from the transcribed strand of the mammalian DHFR gene;Mellon;Cell,1987

2. DNA repair in an active gene: removal of pyrimidine dimers from the DHFR gene of CHO cells is much more efficient that in the genome overall;Bohr;Cell,1985

3. Failure of RNA synthesis to recover after UV irradiation: an early defect in cells from individuals with Cockayne's syndrome and xeroderma pigmentosum;Mayne;Cancer Res.,1982

4. The residual repair capacity of xeroderma pigmentosum complementation group C fibroblasts is highly specific for transcriptionally active DNA;Venema;Proc. Natl. Acad. Sci. U.S.A.,1990

5. Molecular cloning of the human DNA excision repair gene ERCC-6;Troelstra;Mol. Cell. Biol.,1990

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