Beneficial effects of SR33805 in failing myocardium-Reference-Cited by-同舟云学术

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Beneficial effects of SR33805 in failing myocardium

Published:2011-04-04 Issue:3 Volume:91 Page:412-419
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Ait Mou Younss,Toth Attila,Cassan Cécile,Czuriga Daniel,de Tombe Pieter P.,Papp Zoltan,Lacampagne Alain,Cazorla Olivier

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/91/3/412/17199963/cvr096.pdf

Reference42 articles.

1. Acute heart failure: inotropic agents and their clinical uses;Endoh;Expert Opin Pharmacother,2006

2. Mechanism of action of Ca2+ sensitizers–update 2001;Endoh;Cardiovasc Drugs Ther,2001

3. The contractile apparatus as a target for drugs against heart failure: interaction of levosimendan, a calcium sensitiser, with cardiac troponin c;Sorsa;Mol Cell Biochem,2004

4. SR33805, a Ca2+ antagonist with length-dependent Ca2+ -sensitizing properties in cardiac myocytes;Cazorla;Br J Pharmacol,2003

5. Characterization of the binding of [3H]SR 33805 to the slow Ca2+ channel in rat heart sarcolemmal membrane;Chatelain;Eur J Pharmacol,1994

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