Enhanced susceptibility to biomechanical stress in ACE2 null mice is prevented by loss of the p47phox NADPH oxidase subunit-Reference-Cited by-同舟云学术

Enhanced susceptibility to biomechanical stress in ACE2 null mice is prevented by loss of the p47phox NADPH oxidase subunit

Published:2011-02-01 Issue:1 Volume:91 Page:151-161
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Bodiga Sreedhar,Zhong Jiu Chang,Wang Wang,Basu Ratnadeep,Lo Jennifer,Liu George C.,Guo Danny,Holland Steven M.,Scholey James W.,Penninger Josef M.,Kassiri Zamaneh,Oudit Gavin Y.

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/91/1/151/17391991/cvr036.pdf

Reference44 articles.

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2. Gain-of-function mutant of angiotensin II receptor, type 1A, causes hypertension and cardiovascular fibrosis in mice;Billet;J Clin Invest,2007

3. Angiotensin II stimulates NADH and NADPH oxidase activity in cultured vascular smooth muscle cells;Griendling;Circ Res,1994

4. NAD(P)H oxidase: role in cardiovascular biology and disease;Griendling;Circ Res,2000

5. Role of p47(phox) in vascular oxidative stress and hypertension caused by angiotensin II;Landmesser;Hypertension,2002

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