Physiological and pathological ventricular hypertrophy: a role for cavin-2 mediated Akt regulation
Author:
Affiliation:
1. Department NeuroFarBa, University of Florence , Viale G. Pieraccini 6, 50139 Florence , Italy
Publisher
Oxford University Press (OUP)
Link
https://academic.oup.com/cardiovascres/advance-article-pdf/doi/10.1093/cvr/cvae184/59028486/cvae184.pdf
Reference10 articles.
1. Evolution of ventricular hypertrophy and myocardial mechanics in physiological and pathological hypertrophy;Yalcin;J Appl Physiol (1985),2019
2. Late sodium current inhibition reverses electromechanical dysfunction in human hypertrophic cardiomyopathy;Coppini;Circulation,2013
3. Phosphoinositide 3-kinase(p110alpha) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy;McMullen;Proc Natl Acad Sci U S A,2003
4. AKT2 blocks nucleus translocation of apoptosis-inducing factor (AIF) and endonuclease G (EndoG) while promoting caspase activation during cardiac ischemia;Yang;Int J Mol Sci,2017
5. Protective effects of exercise and phosphoinositide 3-kinase(p110alpha) signaling in dilated and hypertrophic cardiomyopathy;McMullen;Proc Natl Acad Sci U S A,2007
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