Peanut lectin stimulates proliferation of colon cancer cells by interaction with glycosylated CD44v6 isoforms and consequential activation of c-Met and MAPK: functional implications for disease-associated glycosylation changes
Author:
Publisher
Oxford University Press (OUP)
Subject
Biochemistry
Link
http://academic.oup.com/glycob/article-pdf/16/7/594/1637434/cwj108.pdf
Reference44 articles.
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3. Boland, C.R. and Roberts, J.A. (1988) Quantitation of lectin binding sites in human colon mucins by use of peanut and wheat germ agglutinins. J. Histochem. Cytochem., 36, 1305–1307.
4. Campbell, B.J. , Finnie, I.A., Hounsell, E.F., and Rhodes, J.M. (1995) Direct demonstration of increased expression of Thomsen-Friedenreich (TF) antigen in colonic adenocarcinoma and ulcerative colitis mucin and its concealment in normal mucin. J. Clin. Invest., 95, 571–576.
5. Campbell, B.J. , Yu, L.G., and Rhodes, J.M. (2001) Altered glycosylation in inflammatory bowel disease: a possible role in cancer development. Glycoconj. J., 18, 851–858.
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