Inhibition of clonal expansion of parietal epithelial cells and crescent–podocyte transition in severe glomerulonephritis: on the way to targeted therapy?
Author:
Affiliation:
1. Department of Nephrology, Marmara University School of Medicine , Istanbul , Turkey
2. Department of Medicine, University of Cambridge , Cambridge , UK
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
https://academic.oup.com/ckj/advance-article-pdf/doi/10.1093/ckj/sfad054/50404686/sfad054.pdf
Reference10 articles.
1. Characterization of renal progenitors committed toward tubular lineage and their regenerative potential in renal tubular injury;Angelotti;Stem Cells,2012
2. Podocyte regeneration driven by renal progenitors determines glomerular disease remission and can be pharmacologically enhanced;Lasagni;Stem Cell Reports,2015
3. Differentiation of crescent-forming kidney progenitor cells into podocytes attenuates severe glomerulonephritis in mice;Melica;Sci Transl Med,2022
4. CXCL12 blockade preferentially regenerates lost podocytes in cortical nephrons by targeting an intrinsic podocyte-progenitor feedback mechanism;Romoli;Kidney Int,2018
5. Therapeutic inhibition of VEGF signaling and associated nephrotoxicities;Estrada;J Am Soc Nephrol,2019
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