Preventing AID, a physiological mutator, from deleterious activation: regulation of the genomic instability that is associated with antibody diversity
Author:
Publisher
Oxford University Press (OUP)
Subject
Immunology,General Medicine,Immunology and Allergy
Link
http://academic.oup.com/intimm/article-pdf/22/4/227/2641975/dxq023.pdf
Reference121 articles.
1. Molecular Mechanism of Class Switch Recombination: Linkage with Somatic Hypermutation
2. Mini-review Activation-induced cytidine deaminase: a dual role in class-switch recombination and somatic hypermutation
3. Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2)
4. Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme
5. AID Enzyme-Induced Hypermutation in an Actively Transcribed Gene in Fibroblasts
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