Improvement of BDNF signalling by P42 peptide in Huntington's disease-Reference-Cited by-同舟云学术

Improvement of BDNF signalling by P42 peptide in Huntington's disease

Published:2018-06-01 Issue:17 Volume:27 Page:3012-3028
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Couly Simon¹,Paucard Alexia¹,Bonneaud Nathalie¹,Maurice Tangui¹,Benigno Lorraine²,Jourdan Christophe¹,Cohen-Solal Catherine³,Vignes Michel³,Maschat Florence¹

Affiliation:

1. MMDN, Univ-Montpellier, EPHE, INSERM, UMR-S1198, Montpellier F-34095, France

2. Medesis Pharma, Baillargues F-34670, France

3. IBMM-UMR5247, Univ-Montpellier, Montpellier F-34095, France

Funder

Agence Nationale de la Recherche

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/27/17/3012/25505594/ddy207.pdf

Reference65 articles.

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2. The P42 peptide and peptide-based therapies for Huntington's disease;Marelli;Orphanet. J. Rare Dis,2016

3. The prevalence of Huntington's disease;Rawlins;Neuroepidemiology,2016

4. Caspase 3-cleaved N-terminal fragments of wild-type and mutant huntingtin are present in normal and Huntington's disease brains, associate with membranes, and undergo calpain-dependent proteolysis;Kim;Proc. Natl. Acad. Sci. U. S. A,2001

5. Caspase cleavage of mutant huntingtin precedes neurodegeneration in Huntington's disease;Wellington;J. Neurosci,2002

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