Tumor Necrosis Factor-alpha Potentiates the Cytotoxicity of Amiodarone in Hepa1c1c7 Cells: Roles of Caspase Activation and Oxidative Stress

Author:

Lu Jingtao,Miyakawa Kazuhisa,Roth Robert A.,Ganey Patricia E.

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Reference56 articles.

1. Signalling pathways involved in 1-nitropyrene (1-NP)-induced and 3-nitrofluoranthene (3-NF)-induced cell death in Hepa1c1c7 cells;Mutagenesis,(2009)

2. Amiodarone hepatotoxicity;Curr. Vasc. Pharmacol.,(2008)

3. Amiodarone induces apoptosis of human and rat alveolar epithelial cells in vitro;Am. J. Physiol. Lung Cell Mol. Physiol.,(2000)

4. Lipopolysaccharide signal transduction, regulation of tumor necrosis factor biosynthesis, and signaling by tumor necrosis factor itself;J. Cardiovasc. Pharmacol.,(1995)

5. Disruption of mitochondrial function and cellular ATP levels by amiodarone and N-desethylamiodarone in initiation of amiodarone-induced pulmonary cytotoxicity;J. Pharmacol. Exp. Ther.,(2001)

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