Molecular basis of intrinsic macrolide resistance in clinical isolates of Mycobacterium fortuitum-Reference-Cited by-同舟云学术

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Molecular basis of intrinsic macrolide resistance in clinical isolates of Mycobacterium fortuitum

Published:2005-02-01 Issue:2 Volume:55 Page:170-177
ISSN:1460-2091
Container-title:Journal of Antimicrobial Chemotherapy
language:en
Short-container-title:

Author:

Nash Kevin A.,Zhang Yansheng,Brown-Elliott Barbara A.,Wallace Richard J.

Publisher

Oxford University Press (OUP)

Subject

Infectious Diseases,Pharmacology (medical),Pharmacology,Microbiology (medical)

Link

http://academic.oup.com/jac/article-pdf/55/2/170/2544474/dkh523.pdf

Reference35 articles.

1. Brown-Elliott, B. A., Griffith, D. E. & Wallace, R. J., Jr (2002). Newly described or emerging human species of nontuberculous mycobacteria. Infectious Disease Clinics of North America16, 187–220.

2. Brown-Elliott, B. A. & Wallace, R. J., Jr (2002). Clinical and taxonomic status of pathogenic nonpigmented or late-pigmenting rapidly growing mycobacteria. Clinical Microbiology Reviews15, 716–46.

3. Bosne-David, S., Barros, V., Verde, S. C. et al. (2000). Intrinsic resistance of Mycobacterium tuberculosis to clarithromycin is effectively reversed by subinhibitory concentrations of cell wall inhibitors. Journal of Antimicrobial Chemotherapy46, 391–5.

4. Luna-Herrera, J., Reddy, V., Daneluzzi, D. et al. (1995). Antituberculosis activity of clarithromycin. Antimicrobial Agents and Chemotherapy39, 2692–5.

5. Garza-Ramos, G., Xiong, L., Zhong, P. et al. (2001). Binding site of macrolide antibiotics on the ribosome: new resistance mutation identifies a specific interaction of ketolides with rRNA. Journal of Bacteriology183, 6898–907.

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