Nicotine Suppresses Tunicamycin-Induced, But Not Thapsigargin-Induced, Expression of GRP78 during ER Stress-Mediated Apoptosis in PC12 Cells-Reference-Cited by-同舟云学术

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Nicotine Suppresses Tunicamycin-Induced, But Not Thapsigargin-Induced, Expression of GRP78 during ER Stress-Mediated Apoptosis in PC12 Cells

Published:2008-04-04 Issue:2 Volume:144 Page:251-257
ISSN:0021-924X
Container-title:Journal of Biochemistry
language:en
Short-container-title:Journal of Biochemistry

Author:

Sasaya H.,Utsumi T.,Shimoke K.,Nakayama H.,Matsumura Y.,Fukunaga K.,Ikeuchi T.

Publisher

Oxford University Press (OUP)

Subject

Molecular Biology,Biochemistry,General Medicine

Link

http://academic.oup.com/jb/article-pdf/144/2/251/6442645/mvn063.pdf

Reference27 articles.

1. The Role of Neuronal Nicotinic Acetylcholine Receptors in Acute and Chronic Neurodegeneration

2. Nicotinic acetylcholine receptors and the regulation of neuronal signalling

3. Activation of α 7 nicotinic acetylcholine receptor promotes survival of spinal cord motoneurons

4. Nicotinic cholinergic stimulation promotes survival and reduces motility of cultured rat cerebellar granule cells

5. Stimulation of α4β2 nicotinic acetylcholine receptors inhibits β-amyloid toxicity

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