Apamin Improves Prefrontal Nicotinic Impairment in Mouse Model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

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Apamin Improves Prefrontal Nicotinic Impairment in Mouse Model of Alzheimer’s Disease

Published:2019-06-12 Issue: Volume: Page:
ISSN:1047-3211
Container-title:Cerebral Cortex
language:en
Short-container-title:

Author:

Proulx É¹,Power S K¹,Oliver D K¹,Sargin D¹,McLaurin J²³,Lambe E K¹⁴⁵^ORCID

Affiliation:

1. Department of Physiology, University of Toronto, Toronto, Ontario, Canada, M5S 1A8

2. Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Ontario, Canada M5S 1A8

3. Biological Sciences and Hurvitz Brain Sciences Research Program, Sunnybrook Research Institute, Toronto, Ontario, Canada M4N 3M5

4. Department of Obstetrics and Gynaecology, University of Toronto, Toronto, Ontario, Canada M5G 1E2

5. Department of Psychiatry, University of Toronto, Toronto, Ontario, Canada M5T 1R8

Funder

Canadian Institutes of Health Research

Canada Research Chair in Developmental Cortical Physiology

Ontario Mental Health Foundation

Publisher

Oxford University Press (OUP)

Subject

Cellular and Molecular Neuroscience,Cognitive Neuroscience

Link

http://academic.oup.com/cercor/advance-article-pdf/doi/10.1093/cercor/bhz107/28796069/bhz107.pdf

Reference65 articles.

1. Developmental sex differences in nicotinic currents of prefrontal layer VI neurons in mice and rats;Alves;PLoS One.,2010

2. Attentional control in Alzheimer's disease;Baddeley;Brain.,2001

3. The nicotinic acetylcholine receptor alpha5 subunit plays a key role in attention circuitry and accuracy;Bailey;J Neurosci.,2010

4. Nicotinic α5 subunits drive developmental changes in the activation and morphology of prefrontal cortex layer VI neurons;Bailey;Biol Psychiatry.,2012

5. Small conductance Ca2+−activated K+ channel knock-out mice reveal the identity of calcium-dependent afterhyperpolarization currents;Bond;J Neurosci.,2004

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