Identification of ter94, Drosophila VCP, as a strong modulator of motor neuron degeneration induced by knockdown of Caz, Drosophila FUS-Reference-Cited by-同舟云学术

Identification of ter94, Drosophila VCP, as a strong modulator of motor neuron degeneration induced by knockdown of Caz, Drosophila FUS

Published:2014-02-04 Issue:13 Volume:23 Page:3467-3480
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Azuma Yumiko,Tokuda Takahiko,Shimamura Mai,Kyotani Akane,Sasayama Hiroshi,Yoshida Tomokatsu,Mizuta Ikuko,Mizuno Toshiki,Nakagawa Masanori,Fujikake Nobuhiro,Ueyama Morio,Nagai Yoshitaka,Yamaguchi Masamitsu

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/23/13/3467/19638018/ddu055.pdf

Reference50 articles.

1. ALS: a disease of motor neurons and their nonneuronal neighbors;Boillée;Neuron,2006

2. TDP-43 and FUS in amyotrophic lateral sclerosis and frontotemporal dementia;Mackenzie;Lancet Neurol.,2010

3. The overlap of amyotrophic lateral sclerosis and frontotemporal dementia;Lomen-Hoerth;Neurology,2002

4. Continuum of frontal lobe impairment in amyotrophic lateral sclerosis;Murphy;Arch. Neurol.,2007

5. Fused in sarcoma (FUS): an oncogene goes awry in neurodegeneration;Dormann;Mol. Cell Neurosci.,2013

Cited by 37 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Caspar determines primordial germ cell identity in Drosophila melanogaster;2024-06-21

2. Caspar determines primordial germ cell identity in Drosophila melanogaster;2024-06-21

3. Caspar specifies primordial germ cell count and identity inDrosophila melanogaster;2024-04-28

4. Valosin containing protein (VCP): initiator, modifier, and potential drug target for neurodegenerative diseases;Molecular Neurodegeneration;2023-08-07

5. Caspar, an adapter for VAPB and TER94, modulates the progression of ALS8 by regulating IMD/NFκB-mediated glial inflammation in a Drosophila model of human disease;Human Molecular Genetics;2022-04-04