Editorial: Switching on arginase in M2 macrophages
Author:
Funder
National Institutes of Health
Publisher
Wiley
Subject
Cell Biology,Immunology,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1189/jlb.0411203/fullpdf
Reference13 articles.
1. Interleukin 4 potently enhances murine macrophage mannose receptor activity: a marker of alternative immunologic macrophage activation;Stein;J. Exp. Med.,1992
2. Differential regulation of nitric oxide synthase-2 and arginase-1 by type 1/type 2 cytokines in vivo: granulomatous pathology is shaped by the pattern of L-arginine metabolism;Hesse;J. Immunol.,2001
3. Alternative activation is an innate response to injury that requires CD4 + T cells to be sustained during chronic infection;Loke;J. Immunol.,2007
4. Suppressor of cytokine signaling (SOCS)1 is a key determinant of differential macrophage activation and function
5. Suppressor of cytokine signaling-1 is an IL-4-inducible gene in macrophages and feedback inhibits IL-4 signaling;Dickensheets;Genes Immun.,2007
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