Lipid phosphate phosphatase 3 enables efficient thymic egress

Author:

Bréart Béatrice1,Ramos-Perez Willy D.1,Mendoza Alejandra1,Salous Abdelghaffar K.2,Gobert Michael1,Huang Yong3,Adams Ralf H.45,Lafaille Juan J.1,Escalante-Alcalde Diana6,Morris Andrew J.2,Schwab Susan R.1

Affiliation:

1. Program in Molecular Pathogenesis and Department of Pathology, Skirball Institute of Biomolecular Medicine, New York University School of Medicine, New York, NY 10016

2. Division of Cardiovascular Medicine, Gill Heart Institute, University of Kentucky, Lexington, KY 40536

3. Department of Bioengineering and Therapeutic Sciences, University of California, San Francisco, South San Francisco, CA 94080

4. Department of Tissue Morphogenesis, Max Planck Institute for Molecular Biomedicine, 48149 Münster, Germany

5. Faculty of Medicine, University of Münster, 48149 Münster, Germany

6. División de Neurociencias, Instituto de Fisiología Celular, Universidad Nacional Autónoma de México, México City 04510, México

Abstract

The signaling lipid sphingosine-1-phosphate (S1P) stabilizes the vasculature, directs lymphocyte egress from lymphoid organs, and shapes inflammatory responses. However, little is known about how S1P distribution is controlled in vivo, and it is not clear how a ubiquitously made lipid functions as a signal that requires precise spatial and temporal control. We have found that lipid phosphate phosphatase 3 (LPP3) enables efficient export of mature T cells from the thymus into circulation, and several lines of evidence suggest that LPP3 promotes exit by destroying thymic S1P. Although five additional S1P-degrading enzymes are expressed in the thymus, they cannot compensate for the loss of LPP3. Moreover, conditional deletion of LPP3 in either epithelial cells or endothelial cells is sufficient to inhibit egress. These results suggest that S1P generation and destruction are tightly regulated and that LPP3 is essential to establish the balance.

Publisher

Rockefeller University Press

Subject

Immunology,Immunology and Allergy

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