Gene-Gene and Gene-Environment Interactions Involving HLA-DRB1, PTPN22, and Smoking in Two Subsets of Rheumatoid Arthritis-Reference-Cited by-同舟云学术

Gene-Gene and Gene-Environment Interactions Involving HLA-DRB1, PTPN22, and Smoking in Two Subsets of Rheumatoid Arthritis

Published:2007-05 Issue:5 Volume:80 Page:867-875
ISSN:0002-9297
Container-title:The American Journal of Human Genetics
language:en
Short-container-title:The American Journal of Human Genetics

Author:

Källberg Henrik,Padyukov Leonid,Plenge Robert M.,Rönnelid Johan,Gregersen Peter K.,van der Helm-van Mil Annette H.M.,Toes Rene E.M.,Huizinga Tom W.,Klareskog Lars,Alfredsson Lars

Publisher

Elsevier BV

Subject

Genetics (clinical),Genetics

Reference27 articles.

1. The shared epitope hypothesis;Gregersen;Arthritis Rheum,1987

2. Refining the complex rheumatoid arthritis phenotype based on specificity of the HLA-DRB1 shared epitope for antibodies to citrullinated proteins;Huizinga;Arthritis Rheum,2005

3. Replication of putative candidate-gene associations with rheumatoid arthritis in >4,000 samples from North America and Sweden: association of susceptibility with PTPN22, CTLA4, and PADI4;Plenge;Am J Hum Genet,2005

4. A missense single-nucleotide polymorphism in a gene encoding a protein tyrosine phosphatase (PTPN22) is associated with rheumatoid arthritis;Begovich;Am J Hum Genet,2004

5. Quantification of the influence of cigarette smoking on rheumatoid arthritis: results from a population based case-control study, using incident cases;Stolt;Ann Rheum Dis,2003

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