Primary myelofibrosis is not primary anymore since the discovery of MPL515 and CALR mutations as driver causes of mono-linear megakaryocytic and dual megakaryocytic granulocytic myeloproliferation and secondary myelofibrosis
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Publisher
Heighten Science Publications Corporation
Link
https://www.bonemarrowjournal.com/articles/ijbmr-aid1003.pdf
Reference18 articles.
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2. 2. Michiels JJ, Thiele J. Clinical and pathological criteria for the diagnosis of essential thrombocythemia, polycythemia vera and idiopathic myelofibrosis (agnogenic myeloid metaplasia). Int J Hematol. 2002; 76: 133-145. Ref.: http://tinyurl.com/y3r2hp25
3. 3. Tefferi A1, Lasho TL, Jimma T, Finke CM, Gangat N, et al. One thousand patients with primary myelofibrosis: the Mayo Clinici experience. Mayo Clinic Proc. 2012; 87: 25-33. Ref.: http://tinyurl.com/y4eyy6nw
4. 4. Klampf Tl, Gisslinger H, Harutyunyan AS, Nivarthi H, Rumi E, et al. Somatic mutations of calreticulin in myeloproliferative neoplasms. New Eng J Med December. 2013; 369: 2379-2387. Ref.: http://tinyurl.com/y3hc7vte
5. 5. Rumi E, Pietra D, Ferretti V, Klampfl T, Harutyunyan AS, et al. Jak2 or CALR mutation status defines subtypes of essential thrombocythemia with substantially different clinical course and outcome. Blood, 2014; 123: 1552-1565. Ref.: http://tinyurl.com/y4vyyotn
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