A non-cardiomyocyte autonomous mechanism of cardioprotection involving the SLO1 BK channel

Author:

Wojtovich Andrew P.1,Nadtochiy Sergiy M.2,Urciuoli William R.2,Smith Charles O.3,Grunnet Morten45,Nehrke Keith1,Brookes Paul S.2

Affiliation:

1. Department of Medicine, University of Rochester Medical Center, Rochester, NY, USA

2. Department of Anesthesiology, University of Rochester Medical Center, Rochester, NY, USA

3. Department of Biochemistry, University of Rochester Medical Center, Rochester, NY, USA

4. Lundbeck AS, Valby, Denmark

5. Acesion Pharma, Copenhagen, Denmark

Publisher

PeerJ

Subject

General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology,General Medicine,General Neuroscience

Reference69 articles.

1. Mitochondrial matrix K+ flux independent of large-conductance Ca2+-activated K+ channel opening;Aldakkak;American Journal of Physiology – Cell Physiology,2010

2. Energetic performance is improved by specific activation of K+ fluxes through KCa channels in heart mitochondria;Aon;Biochimica et Biophysica Acta,2010

3. Comparative effects of endothelin and neurotensin on intrinsic cardiac neurons in situ;Armour;Peptides,1996

4. Histamine-sensitive intrinsic cardiac and intrathoracic extracardiac neurons influence cardiodynamics;Armour;American Journal of Physiology,1996

5. Intrinsic cardiac neurons involved in cardiac regulation possess alpha 1-, alpha 2-, beta 1- and beta 2-adrenoceptors;Armour;Canadian Journal of Cardiology,1997

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